pancreatic cancer

Research into how pancreatic tumors adapt to stress could lead to a new treatment approach.

This year, cancer researchers uncovered a variety of ways that tumors can survive and spread, ranging from damaging their own DNA to exploiting the nearby microenvironment for nutrients.

Two studies in mice now show that researchers can control when and where CAR T cells are active, potentially overcoming previous hurdles for CAR T–based treatments.

A study reveals a molecular pathway linking exercise to an amped-up immune response to pancreatic cancer and greater responsiveness to treatment.

Because most people are vaccinated against tetanus as children, delivering benign bacteria carrying a tetanus antigen into pancreatic tumors makes them visible to memory cells in the immune system, researchers report.

A key mutation turns healing cells into cancer promoters.

Pancreatic cells associated with cancer resemble celestial bodies as revealed by microscopy.

The test can pick up several types of cancer, including pancreatic and ovarian, years before symptoms appear.

In the largest study of its kind to date, researchers find more than 850 rare, heritable genetic alterations that can predispose humans to cancer.

The new diagnostic works by identifying cancer-associated proteins and DNA in patient blood.

Mouse organoids reveal that a protein active during embryonic development joins forces with gene enhancers to revert cancer cells to an earlier developmental state.

A newly identified marker for a common pancreatic cancer may help doctors detect the disease in its earlier stages.

The mix of bacteria living in the oral cavity is related to a person’s risk of developing pancreatic cancer, according to a study.

Personalized pancreatic cancer therapies based on tumor genomics may take too long to prepare to be helpful, according to a small clinical trial.

Researchers use bacteria to deliver radiation to shrink pancreatic tumors in mice.

What researchers are learning as they sequence, map, and decode species’ genomes

Two proteins interact to save adhesion molecules from degradation, potentially contributing to a more aggressive cancer.